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  Ischemic Heart Disease

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Articles published in Cardiovasc Res

Retrieve available abstracts of 15 articles:
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Single Articles


    November 2017
  1. ZIEGLER KA, Ahles A, Wille T, Kerler J, et al
    Local sympathetic denervation attenuates myocardial inflammation and improves cardiac function after myocardial infarction in mice.
    Cardiovasc Res. 2017 Nov 24. pii: 4657089. doi: 10.1093.
    PubMed     Text format     Abstract available


    September 2017
  2. PARK KC, Gaze DC, Collinson PO, Marber MS, et al
    Cardiac troponins: from myocardial infarction to chronic disease.
    Cardiovasc Res. 2017 Sep 14. doi: 10.1093.
    PubMed     Text format     Abstract available


  3. HAUSENLOY DJ, Garcia-Dorado D, Erik Botker H, Davidson SM, et al
    Melatonin as a cardioprotective therapy following ST-segment elevation myocardial infarction: is it really promising? Reply.
    Cardiovasc Res. 2017;113:1418-1419.
    PubMed     Text format    


    July 2017
  4. BAYOUMI AS, Teoh JP, Aonuma T, Yuan Z, et al
    MicroRNA-532 protects the heart in acute myocardial infarction, and represses prss23, a positive regulator of endothelial-to-mesenchymal transition.
    Cardiovasc Res. 2017 Jul 11. doi: 10.1093.
    PubMed     Text format     Abstract available


    April 2017
  5. WANG Z, Huang S, Sheng Y, Peng X, et al
    Topiramate modulates post-infarction inflammation primarily by targeting monocytes or macrophages.
    Cardiovasc Res. 2017;113:475-487.
    PubMed     Text format     Abstract available


    March 2017
  6. BROMAGE DI, Pickard JM, Rossello X, Ziff OJ, et al
    Remote ischaemic conditioning reduces infarct size in animal in vivo models of ischaemia-reperfusion injury: a systematic review and meta-analysis.
    Cardiovasc Res. 2017;113:288-297.
    PubMed     Text format     Abstract available


    February 2017
  7. LIM S, Lee GY, Park HS, Lee DH, et al
    Attenuation of carotid neointimal formation after direct delivery of a recombinant adenovirus expressing glucagon-like peptide-1 in diabetic rats.
    Cardiovasc Res. 2017;113:183-194.
    PubMed     Text format     Abstract available


    January 2017
  8. JOSE ALBURQUERQUE-BEJAR J, Barba I, Valls-Lacalle L, Ruiz-Meana M, et al
    Remote ischemic conditioning provides humoural cross-species cardioprotection through glycine receptor activation.
    Cardiovasc Res. 2017;113:52-60.
    PubMed     Text format     Abstract available


  9. RAWAL S, Munasinghe PE, Shindikar A, Paulin J, et al
    Down-regulation of proangiogenic microRNA-126 and microRNA-132 are early modulators of diabetic cardiac microangiopathy.
    Cardiovasc Res. 2017;113:90-101.
    PubMed     Text format     Abstract available


    November 2016
  10. LI L, Li J, Drum BM, Chen Y, et al
    Loss of AKAP150 promotes pathological remodelling and heart failure propensity by disrupting calcium cycling and contractile reserve.
    Cardiovasc Res. 2016.
    PubMed     Text format     Abstract available


    July 2016
  11. TAKADA S, Masaki Y, Kinugawa S, Matsumoto J, et al
    Dipeptidyl peptidase-4 inhibitor improved exercise capacity and mitochondrial biogenesis in mice with heart failure via activation of GLP-1 receptor signaling.
    Cardiovasc Res. 2016.
    PubMed     Text format     Abstract available


    May 2016
  12. LEMCKERT FA, Bournazos A, Eckert DM, Kenzler M, et al
    Lack of MG53 in human heart precludes utility as a biomarker of myocardial injury or endogenous cardioprotective factor.
    Cardiovasc Res. 2016;110:178-87.
    PubMed     Text format     Abstract available


  13. EERENBERG ES, Teunissen PF, van den Born BJ, Meijers JC, et al
    The role of ADAMTS13 in acute myocardial infarction: cause or consequence?
    Cardiovasc Res. 2016.
    PubMed     Text format     Abstract available


    February 2016
  14. TRITTO I, Ambrosio G
    Why does pre-clinical success in cardioprotection fail at the bedside?
    Cardiovasc Res. 2016;109:189-90.
    PubMed     Text format    


    January 2016
  15. GONZALEZ-SANTAMARIA J, Villalba M, Busnadiego O, Lopez-Olaneta MM, et al
    Matrix cross-linking lysyl oxidases are induced in response to myocardial infarction and promote cardiac dysfunction.
    Cardiovasc Res. 2016;109:67-78.
    PubMed     Text format     Abstract available


Thank you for your interest in scientific medicine.


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